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This book is about the p53 gene, one of the most frequently mutated or deleted genes in human cancers. The frequent occurrence;
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cases where mutations in only one allele will cause an effect. A notable example is the gene that codes for p53. Tumor suppressor genes, or more;
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contains more than 80 entries on oncogenes including JUN, MYC, and RAS, as well as DNA tumour viruses, tumour suppressor genes, including p53;
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The p53 tumor suppressor gene---"the "guardian of the genome"--- protects cells against genotoxic stress but is mutated in many cancers. It;
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radiation sensitization in clinical practice. The transcription and translation of a set of genes is being restrained by a tumor suppressor gene i.e;
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control; the retinoblastoma gene product and its relatives; the tumour suppressor gene p53; tumour suppressors and the inheritance of cancer; the;
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therapy. PKC is implicated in a transcriptional regulation of the p53 tumor suppressor that is critical for cell cycle arrest and apoptosis;
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Contains papers and discussion from the June 1995 symposium, covering oncogene and tumor suppressor gene function, transcription regulation;
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-53 suppressor gene and oncogene, hematopoietic stem cells, cytokines, detection of minimal residual disease in acute lymphoblastic leukemia;
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P53 ALS Prognostischer Marker Fur Hamatologische Neoplasien is een boek van Nina Obertopp;
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oral cancer; tumour suppressor gene detection in oral tumours for new therapeutic strategy; innate immunity and oral carcinogenesis and;
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and RAF, and the role of tumor suppressors like p53, are discussed in detail. The volume also explores the emerging role of noncoding RNAs;
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of growth factors in the regulation of proto-oncogene and tumor suppressor gene expression, and the development of neoplastic processes is discussed;
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of growth factors in the regulation of proto-oncogene and tumor suppressor gene expression, and the development of neoplastic processes is discussed;
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chromosomal instability in cancer disrupts gene function by either inactivating tumor suppressor genes or activating growth-promoting oncogenes. The;
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carcinogenesis. Recently, Human Papillomavirus (HPV) infection and p53 gene mutation has been shown to play a role in the development of oral cancer;
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comprehensive assessment of the field including the areas of suicide gene therapy, oncogene and suppressor gene targeting, immunotherapy, drug;
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characteristics which are important for each stage. Recently, a class of genes known as metastasis suppressors' has been the subject of intense;
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volume presents outstanding and original reviews on a variety of topics, including suppressor and oncogenic roles of TGF-b and its signaling;
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Inflammatory markers, oncogenes, tumor suppressor genes and cell cycle regulators in skin growths represent a series of reports and reviews;
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of renal cell carcinoma occurring with von Hippel-Landau syndrome; summarizes efforts to identify the tumour suppressor gene deleted from;
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, kidney, and lung; melanoma; HIV; and coronary restenosis includes examples of antisense, ribozyme, tumor suppressor, immunostimulation, and;
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The Von Hippel-Lindau Tumor Suppressor Complex and Regulation of Hypoxia -Inducible Transcription. Retinoblastoma Tumor Suppressor and;
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