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p53 Suppressor Gene

This book is about the p53 gene, one of the most frequently mutated or deleted genes in human cancers. The frequent occurrence;

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p53

p53: Structure, Functions and Role in Disease discusses the role of p53 dysregulation in different hematologic neoplasms defined by the;

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The p53 Family

The p53 tumor suppressor gene---"the "guardian of the genome"--- protects cells against genotoxic stress but is mutated in many cancers. It;

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P53 als prognostischer Marker fur hamatologische Neoplasien

P53 ALS Prognostischer Marker Fur Hamatologische Neoplasien is een boek van Nina Obertopp;

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Clinical Aspects of Radiation Oncology

. P53; and its metamorphosis has been involved in 60% of human cancers. The oscillation of DNA double strand break era and their interdependence;

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Protein Kinase C

drug resistance. A final review is provided which dissects the crosstalk between p53 and PKC in the context of apoptotic cell death and cancer;

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Role of Human Papillomavirus Infection and P53 Mutation in Oral Cancer

carcinogenesis. Recently, Human Papillomavirus (HPV) infection and p53 gene mutation has been shown to play a role in the development of oral cancer;

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p53

. The discovery of p53 using the tumor causing animal virus SV40 falls into this pioneering period of biological and medical research.;

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Sumoylation

, viruses and the tumor suppressor P53.;

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Prognostic and Predictive Factors in Breast Cancer

examines the roles of major predictive markers such as the steroid receptors, p53 and HER-2. Given the wealth of information in the medical;

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International Review of Cytology

Regulation of Hypothalamic-Pituitary-Adrenal Axis; Focal Adhesion and p53 Signaling in Cancer Cells; Cell and Molecular Biology of the Spindle Matrix;

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SIRT6 Activities in DNA Damage Repair and Premature Aging

presents research on the nuclear lamin A, notably the upregulation of p53 and acetylation etc. Taken together, these studies reveal the various;

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Advances in Cancer Research

Implications of the Erb/HER Signaling Network of Growth Factor Receptors p53 and Human Cancer: The First Ten Thousand Mutations

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Role of Capsaicin in Oxidative Stress and Cancer

; identification of p53 activation as a possible mechanism; involvement of Cox-2 in apoptosis, suppression of transcription factors such as NF-kB and STAT;

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Cell Cycle Checkpoints

in this book cover topics such as post-translationally modified p53 by western blotting; CHK1 cellular localization by immunofluorescence;

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The Oncogene and Tumour Suppressor Gene Factsbook

contains more than 80 entries on oncogenes including JUN, MYC, and RAS, as well as DNA tumour viruses, tumour suppressor genes, including p53;

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Approaches to Understanding Breast Cancer

Experimental Therapy of Advanced Breast Cancer: Target NFAT1-MDM2-p53 Pathway. Accessible to students and researchers alike

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Protein-Protein Interactions

Kumar Targeting the MDM2-p53 Protein-Protein Interaction for New Cancer Therapeutics Kurt Deshayes , Jeremy Murray , Domagoj Vucic The;

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Viruses Genes and Cancer

and RAF, and the role of tumor suppressors like p53, are discussed in detail. The volume also explores the emerging role of noncoding RNAs;

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Sphingolipids in Cancer

signaling and p53 pathways, Sphingolipid regulation of RNA Biology in cancer phenotypes, The role of ceramide-1-phosphate in tumor cell survival and;

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Strahlentherapie. Arten und Effekte auf Zellen

der Tumorzellen verschlechtert, beziehungsweise die Kontrollpunkte im Zellzyklus ausser Kraft gesetzt werden. Hierfur ist das p53-Protein verantwortlich;

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Tumor Suppressor Genes

cases where mutations in only one allele will cause an effect. A notable example is the gene that codes for p53. Tumor suppressor genes, or more;

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Allostery

transduction of signals across an organized network structure; and the P53 protein located at the cross-road of cell cycle regulation, genome integrity;

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MiRNAs in Differentiation and Development

damage response by miRNAs, miRNAs and the p53 network, the Impact of miRNAs in the cellular response to hypoxia, miRNAs in oncogenesis and tumor;

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Protein-Protein Interactions In Drug Discovery

* SH3 Domains as Drug Targets * P53 MDM2 Antagonists: Towards Non Genotoxic Anticancer Treatments * Inhibition of LFA-1/ICAM interaction for;

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Oncogenes and Tumour Suppressors

control; the retinoblastoma gene product and its relatives; the tumour suppressor gene p53; tumour suppressors and the inheritance of cancer; the;

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Myelodysplastic Syndrome

+/- is a component of a multiprotein I(2)-catenin destruction complex that regulates Wnt/I(2)-catenin and p53 pathways. The concluding study;

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