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Tumor Necrosis Factor

Tumour Necrosis Factor (TNF) is a member of a superfamily of proteins, each with 157 amino acids, which induce necrosis (death) of tumour;

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Growth, Proliferation, and Apoptosis of Hepatocytes

experimental results. Focusing on apoptosis, guest speaker Professor D.A. Brenner, USA, spoke on the regulation of tumor necrosis factor alpha and Fas;

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Advanced Imaging Techniques in Brain Tumors, An Issue of Neuroimaging Clinics

in brain tumor research; Brain irradiation - effects on normal brain parenchyma and radiation necrosis; MR perfusion and permeability in high-grade;

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Neurological Disorders

associated with anti-tumor necrosis factor alpha (TNF-a) treatment; effect of aging on changes in nociception and Cav2a1 expression patterns;

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Evaluation of Brain Tumor Recurrence- Role of Pet, Spect, MR

high rate of recurrence.There is always some short of confusion in detecting residual or recurrence tumor in the midst of radiation necrosis;

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Tonsillar Disorders

; post-tonsillectomy hemorrhage; peritonsillar abscess and tumour necrosis factor producing cells in chronic tonsillitis.;

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Resistin

apparent that adipose-secreted factors such as leptin, tumor necrosis factor-a, IL-6, adiponectin and more recently, resistin are implicated;

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Tumor Microenvironment Regulation of Tumor Expansion

mainly depends on the release of specific growth factors by inflammatory or neoplastic cells. Inflammatory infiltrate contributes to tumor;

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Landmarking and Segmentation of 3D CT Images

neuroblastic tumor, ranging from low-attenuation necrosis to high-attenuation calcification, segmentation of the tumor mass is a challenging problem;

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Hypoxia & Cancer

feature of the tumor microenvironment. Hypoxia Inducible Factor 1 (HIF-1) is a master regulator of the transcriptional response to oxygen;

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Necrotic Cell Death

Starting with discussion of basic concepts and the molecular mechanisms of necrosis, this book looks first at several forms of necrotic;

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Avascular necrosis of the foot and ankle, An issue of Foot and Ankle Clinics of North America

This issue of Foot and Ankle Clinics, guest edited by Dr. Kenneth Hunt, will cover concepts surrounding Avascular Necrosis of the Foot and;

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Apoptosis and Beyond

apoptotic pathway. In addition, it links activities like the molecular biology of one process (eg. Necrosis) to another process (eg. apoptosis) and;

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Neuroradiology, An Issue of Radiologic Clinics of North America

. Articles will include: CT Perfusion in Acute Stroke; The Role of DTI and fMRI Prior to Brain Tumor Surgery; Pediatric Primary Neoplasms; Radiomics;

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Lipid A in Cancer Therapy

necrosis factor (TNF-a). They do so by engagement of a rather complex set of interactions culminating in the ligation of a Toll-like receptor, TLR;

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Skin Necrosis

involved in wound healing facilitates efficient assessment and treatment of patients with wounds, and skin necrosis can be considered the starting;

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Processing of VEGF-C and -D by the Proprotein Convertases

The vascular endothelial growth factor (VEGF) family members that include VEGF-A, -B, -C, -D, and placental growth factor (PlGF), display;

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Myeloid Cells in Health and Disease

repair of damaged tissue * the vast arsenal of myeloid cell secretory molecules, including metalloproteinases, tumor necrosis factor;

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DNA Tumor Viruses

factor (after tobacco consumption) for cancer development in humans. Today, five virus types are known to be involved in causing human cancer;

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Tumor Immunology and Immunotherapy - Integrated Methods Part B

Tumor Immunology and Immunotherapy - Integrated Methods Part B, Volume 636 in the Methods in Enzymology series, continues the legacy;

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Landmarking and Segmentation of 3D CT Images

neuroblastic tumor, ranging from low-attenuation necrosis to high-attenuation calcification, segmentation of the tumor mass is a challenging problem;

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Beyond Apoptosis

overview of how different biological mechanisms of cell death, senescence and mitotic catastrophe stop the growth of tumor cells treated with;

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Beyond Apoptosis

overview of how different biological mechanisms of cell death, senescence and mitotic catastrophe stop the growth of tumor cells treated with;

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