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book will supply information regarding this pattern in brain cancer.;
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development and integrates the function of tumor suppressor genes, oncogenes, and metastasis-associated genes in the pathogenesis and progression;
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in tumor immunotherapy. In addition, it examines the effect of epigenetic modifications of autophagy-associated genes in regulating tumor growth;
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chromosomal instability in cancer disrupts gene function by either inactivating tumor suppressor genes or activating growth-promoting oncogenes. The;
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The epigenetic regulation plays an important role in normal development and maintenance of tissue specific genes expression in humans and;
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of transcription factors have been associated with cancer. This volume gives molecular information on several oncogenes, tumor suppressor genes or;
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., hedgehog) facilitates targeted therapies in cancer. Tumor models are also presented, which utilize expression data, pathway sensitivity, and;
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Tumour markers are molecules occurring in blood or tissue that are associated with cancer, and whose measurement or identification;
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only tumor initiation but tumor recurrence and patient relapse. The BTSC model recapitulates scientific observations made in brain cancer;
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Contains papers and discussion from the June 1995 symposium, covering oncogene and tumor suppressor gene function, transcription regulation;
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, or tumor suppressor genes - in the etiology of human cancer. Oncogenic signaling by PI3 kinase, mTOR, Akt, or the major cancer drivers MYC;
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Tumor Suppressing Viruses, Genes, and Drugs profiles the new generation of cancer treatments now in development. The book examines;
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levels (hypoxia and/or anoxia) in tumor tissues. Intra-tumor hypoxia profoundly affects the biological behavior of cancer cells, which become;
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development of novel therapeutic strategies in cancer management. This book focuses on metastasis-associated genes, metastasis promoter and suppressor;
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describe the use of differential display to identify novel cancer-associated genes. This is followed by a chapter in which Aronow et al. describe;
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genes associated with progression of malignant behavior in pancreatic cancer. As a result, alterations in the expression of several cancer;
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of methodologies to identify epigenetic marker genes that can be used for the diagnosis as well as for the targeted treatment of tumors. The newly;
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of genetic alterations in cancer-causing genes, such as the oncogenes and the tumor-s- pressor genes. In the last decade, major progress has been;
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available for assessing the diagnosis and prognosis of cancer patients including data on the application of tumor marker assays and other;
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is profoundly involved in the etiology of a wide range of cancers. MYC encodes a transcriptional regulator that modulates expression of genes;
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offers comprehensive analyses of hereditary breast cancer-associated syndromes, low penetrance genes, and hereditary breast cancer associated;
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offers comprehensive analyses of hereditary breast cancer-associated syndromes, low penetrance genes, and hereditary breast cancer associated;
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discovered that for many of these sporadic' (non-familial) cancer cases, defects or aberrations in certain metabolic genes not previously associated;
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Cancer is initiated by activation of oncogenes or inactivation of tumor suppressor genes. Mutations in the K-ras proto-oncogene are;
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research on the role of chemokines in the tumor microenvironment. Each chapter focuses on the chemokines patterns of expression, their regulation;
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research on the role of chemokines in the tumor microenvironment. Each chapter focuses on the chemokines patterns of expression, their regulation;
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