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in the etiology of hypertension. Structural changes of the blood vessels in hypertension in relation to connective tissue, cerebral vessel;
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practitioner, the focus of this volume is on various therapies and how to lower blood pressure through lifestyle changes. Specialist patient;
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, mathematicians, biologists. Topics discussed include a review on genetic and physiological mechanisms of arterial hypertension; descriptions of various;
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The biomolecular basis underlying essential hypertension and end organ damage associated with hypertension is characterized as polygenic;
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blood pressure. Lack of exercise, obesity, high salt intake and depression contribute to hypertension. Various clinical conditions may also;
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narrowing of the vessels increases the pressure within the vessels causing high blood pressure (hypertension). Angiotensin II is formed from;
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vessels. Dysfunction of vascular smooth muscle cells is an important cause of vascular diseases, for example, atherosclerosis, hypertension, and;
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pressure in clinical hypertension. Heart rate, pulse pressure and arterial stiffness. The relation of large arterial structure and function to;
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, water retention, and arterial pressure increase, i.e. a process very similar to development of essential hypertension, which from this;
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This book provides a comprehensive review of the structure, function and pathophysiology of the pulmonary vasculature. Emerging evidence;
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of Heart Failure, Evolution from Hypertension to Heart Failure, Changes in Kidney Function in Heart Failure, Implication of Acute Kidney Injury;
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clearly showed that the large arteries are largely involved in the mechanisms of hypertension not only through changes in mean blood flow, but;
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elicits an increase in systolic blood pressure; poor glucose control and stiff arteries; blood pressure changes in patients with migraines;
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-pharmacological therapies and how to lower blood pressure through lifestyle changes. Specialist patient groups and hypertension during pregnancy are also;
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In the vast majority of patients, hypertension has no identifiable cause. Moreover, despite decades of discovery and therapeutic;
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attacks. This fully revised and updated edition of the ABC of Hypertension is an established guide providing all the non-specialist needs;
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. Excessive salt in the diet, alcohol consumption, smoking and excess body weight are factors that may lead to hypertension. Chronic kidney disease;
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. Hypertension usually does not cause clear identifiable symptoms and hence its identification usually occurs through screening. Physical examination;
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Pulmonary hypertension (PH) is increased pressure in the pulmonary arteries, which carry blood from the heart to the lungs to pick up;
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, as well as medical imaging for pulmonary, cardiac, mammographic applications. Part I includes theories and methods unique to;
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options for each hypertension phenotype. Several of these phenotypes are well known and well described in the literature, such as prehypertension;
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vessels and eventually lead to stroke, heart failure, heart attack, kidney failure and vision problems. Whether you're a newly diagnosed patient;
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layer of specialized endothelial cells endowed with important permeability, transport, and regulatory functions that both protect the internal;
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the American Hypertension Association (AHA), guidelines which lowered the diagnosis thresholds of hypertension, and thereby increased the;
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Pulmonary hypertension is a complex disease that causes significant morbidity and mortality and is clinically characterised by an increase;
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epidemiology and pathophysiology, the book also contains focused reviews on various therapies and how to lower blood pressure through lifestyle changes;
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and physiologic function, maintain tissue homeostasis, and mediate injury response, tissue repair, and regeneration. As a result, FGFs play;
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